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Figure 3-1 Table of examples of microorganisms belonging to the major groups of pathogens
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Figure 3-2 Table of conserved components of microorganisms that are recognized by innate immune cells and molecules
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Figure 3-3 Features of bacterial cell walls that are recognized by components of innate immunity
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Figure 3-4 Table of human antimicrobial peptides and their primary locations
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Figure 3-5 Structural features of defensins
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Figure 3-6 Paneth cells in the small intestine
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Figure 3-7 Mechanism of defensin killing of microbes
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Figure 3-8 Structures of the collectins
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Figure 3-9 MASPs couple MBL and ficolins to the complement pathway
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Figure 3-10 Outline of the complement system
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Figure 3-11 Complement components activated in early and late events in the complement cascade
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Figure 3-12 Table of diseases common in people deficient in complement components
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Figure 3-13 C1q/r/s complex that couples antibody or pentraxin binding to complement activation via the classical pathway
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Figure 3-14 Activation of the complement cascade
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Figure 3-15 Amplification and inactivation of C3b
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Figure 3-16 Late events and effector actions of the complement cascade
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Figure 3-17 Table of complement components and receptors
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Figure 3-18 Structure and function of complement receptors
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Figure 3-19 Mechanisms of action of some complement regulatory proteins
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Figure 3-20 Table of complement regulatory proteins
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Figure 3-21 Microbes and viruses evade complement attack by multiple mechanisms
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Figure 3-22 Mechanisms of particle internalization by phagocytic cells
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Figure 3-23 Table of phagocytic receptors
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Figure 3-24 Different types of phagocytic receptors
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Figure 3-25 Mechanism of phagocytosis
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Figure 3-26 Phagocytosis of apoptotic cells
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Figure 3-27 Table of contents of the primary and secondary granules of neutrophils
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Figure 3-28 Generation of toxic oxygen and nitrogen compounds by phagocytes
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Figure 3-29 Activation-dependent assembly of the NADPH oxidase
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Figure 3-30 Table showing the ligand specificity of mammalian TLRs
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Figure 3-31 Recognition of bacterial lipopolysaccharide by innate immune cells
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Figure 3-32 Cellular localization of TLRs
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Figure 3-33 Two signaling pathways of mammalian TLRs
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Figure 3-34 TLR4 signaling to both the MyD88 pathway and the TRIF pathway
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Figure 3-35 Ligands for NOD1, NOD2 and cryopyrin
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Figure 3-36 Inflammasome assembled in response to muramyl dipeptide
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Figure 3-37 NOD-containing proteins involved in immunity and apoptosis
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Figure 3-38 Innate immune initiation of inflammation
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Figure 3-39 Table of innate immune mediators of inflammation
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Figure 3-40 Arachidonic acid-derived lipid mediators
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Figure 3-41 Recruitment of leukocytes to sites of infection
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Figure 3-42 Table of acute-phase proteins
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Figure 3-43 Positive and negative feedback loops in the production of inflammatory cytokines
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Figure 3-44 Signaling by receptors for interferons
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Figure 3-45 IRFs induce the production of type 1 interferons
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Figure 3-46 Major antiviral actions of interferons-α and β
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Figure 3-47 Evasion of PKR by viruses
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Figure 3-48 Mechanisms of apoptosis induction in virus-infected cells
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Figure 3-49 Signaling by death domain receptors
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Figure 3-50 The conversion catalyzed by cytidine deaminase
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Figure 3-51 Incorporation of APOBEC3G into HIV virions
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Figure 3-52 Mutation of retroviral DNA by APOBEC3G
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Figure 3-53 Biochemical mechanism of RNA interference
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